mouse anti lamin b1 (Santa Cruz Biotechnology)
96
Structured Review
Santa Cruz Biotechnology
mouse anti lamin b1
Mouse Anti Lamin B1, supplied by Santa Cruz Biotechnology, used in various techniques. Bioz Stars score: 96/100, based on 1595 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/lamin+b1/Lamin+B1+Antibody/pm42885327-70-8-13
Average 96 stars, based on 1595 article reviews
Mouse Anti Lamin B1, supplied by Santa Cruz Biotechnology, used in various techniques. Bioz Stars score: 96/100, based on 1595 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/lamin+b1/Lamin+B1+Antibody/pm42885327-70-8-13
Average 96 stars, based on 1595 article reviews
mouse anti lamin b1 - by Bioz Stars,
2026-10
96/100 stars
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Recombinant:Article Title: ENDOU-1-induced cytoplasmic HnRNPA3 recognizes m6A methylation on the upstream reading frame of human CHOP transcripts to achieve maximal CHOP translation Article Snippet: .. The following antibodies were used: CHOP (1:1000; CST, #2895), HnRNPA3 (1:1000; Proteintech, 25142-1-AP), ATF4 (1:1000; Proteintech, 10835-1-AP), eIF2α (1:1,000; CST, #5324); p-eIF2α (1:1,000; CST, #3398); PERK (1:1,000; CST, #3192); p‐PERK (1:1,000; Abcam, Ab156919 ); Bip (1:1,000; Proteintech, 11587-1-AP); Flag (1:20,000; Proteintech, 20543-1-AP); Myc(1:1,000; CST, #2276); ENDOU‐1 (1:1,000; Abcam, EPR15137 ); m6A (1:1000; Proteintech, 68055-1-Ig), METTL14 (1:1,000; Sigma-Aldrich, HPA038002), WTAP (1:1,000; Proteintech, 10200-1-AP), Article Title: ENDOU-1-induced cytoplasmic HnRNPA3 recognizes m6A methylation on the upstream reading frame of human CHOP transcripts to achieve maximal CHOP translation. Article Snippet: .. The following antibodies were used: CHOP (1:1000; CST, #2895), HnRNPA3 (1:1000; Proteintech, 25142-1-AP), ATF4(1:1000; Proteintech, 10835-1-AP), eIF2α (1:1,000; CST, #5324); p‐eIF2α (1:1,000; CST, #3398); PERK (1:1,000; CST, #3192); p‐PERK (1:1,000; Abcam, Ab156919); Bip (1:1,000; Proteintech, 11587-1- AP); Flag (1:20,000; Proteintech, 20543-1-AP); Myc(1:1,000; CST, #2276); ENDOU‐1 (1:1,000; Abcam, EPR15137); m6A (1:1000; Proteintech, 68055-1-Ig), METTL14 (1:1,000; Sigma-Aldrich, HPA038002), WTAP (1:1,000; Proteintech, 10200-1-AP), Marker:Article Title: ENDOU-1-induced cytoplasmic HnRNPA3 recognizes m6A methylation on the upstream reading frame of human CHOP transcripts to achieve maximal CHOP translation. Article Snippet: .. The α-tubulin (1∶10,000; Sigma-Aldrich, T6074) was used as a marker for cytoplasm, and Article Title: ENDOU-1-induced cytoplasmic HnRNPA3 recognizes m6A methylation on the upstream reading frame of human CHOP transcripts to achieve maximal CHOP translation Article Snippet: .. The α-tubulin (1∶10,000; Sigma-Aldrich, T6074) was used as a marker for cytoplasm, and Incubation:Article Title: CIO-IAA is a dual ET A /ET B receptor antagonist that suppresses endothelin-1-induced macrophage activation and inflammatory signaling. Article Snippet: Elevation of endothelin-1 (ET-1), a potent vasoactive and inflammatory peptide, occurs in several cardiovascular and metabolic disorders associated with macrophage activation and inflammation.. Using integrated pharmacological, molecular dynamics simulation (MDS), functional, and cellular approach, we investigated the effects of 2-(6-chloro-1H-indol-3-yl)-2-oxoacetic acid (CIO-IAA) on ET receptor function and ET-1-induced macrophage activation.. CIO-IAA inhibited ET-1-induced reactive oxygen species (ROS) production, NADPH oxidase 2 (Nox2) expression, TNF-α and IL-6 production, Mac-1 (CD11β) expression, macrophage adhesion to endothelial cells, and NF-κB p65 activation and nuclear translocation. Article Title: Electroacupuncture mitigates lung injury, increases endogenous antioxidant levels and modulates Nrf2 signaling in a rat model of chronic obstructive pulmonary disease. Article Snippet: Background: Electroacupuncture (EA) has shown efficacy in the treatment of chronic obstructive pulmonary disease (COPD), but the underlying mechanism of action remains unclear.. Oxidative stress is a main pathological process in COPD.. This aim of this study was to explore the anti-oxidant effect of EA in a COPD rat model. Methods: Rats were divided into control, model, EA, sham acupuncture (SA) and N-acetylcysteine (NAC) groups. Article Title: Tributyl phosphate induces liver aging via fumarate-mediated mitochondrial DNA escape triggered FPR2 activation. Article Snippet: .. Membranes were blocked with 5% non-fat milk in PBS-Tween-20 for 2 h, followed by incubation with primary antibodies against HP1 (Santa Cruz, USA), Cell-Signaling:Article Title: CIO-IAA is a dual ET A /ET B receptor antagonist that suppresses endothelin-1-induced macrophage activation and inflammatory signaling. Article Snippet: Elevation of endothelin-1 (ET-1), a potent vasoactive and inflammatory peptide, occurs in several cardiovascular and metabolic disorders associated with macrophage activation and inflammation.. Using integrated pharmacological, molecular dynamics simulation (MDS), functional, and cellular approach, we investigated the effects of 2-(6-chloro-1H-indol-3-yl)-2-oxoacetic acid (CIO-IAA) on ET receptor function and ET-1-induced macrophage activation.. CIO-IAA inhibited ET-1-induced reactive oxygen species (ROS) production, NADPH oxidase 2 (Nox2) expression, TNF-α and IL-6 production, Mac-1 (CD11β) expression, macrophage adhesion to endothelial cells, and NF-κB p65 activation and nuclear translocation. Binding Assay:Article Title: Oncogenic Signaling Activation and Potential Biomarkers in Congestive Hepatopathy Revealed by Proteomic Analysis. Article Snippet: Aim: Fontan surgery improves outcomes in univentricular heart disease; however, some patients develop severe Fontan‐ associated liver disease (FALD).. Even during adolescence, hepatocellular carcinoma occurs in some patients with FALD, although the mechanism remains unclear.. In this study, we conducted proteomic analyses of liver tissues obtained using laser capture microdissection (LCM) and serum samples to investigate FALD‐related peripheral liver oncogenic factors and explore potential biomarkers. |
